The Journal of Experimental Medicine
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Published online June 30, 2008
doi:10.1084/jem.20072066
The Journal of Experimental Medicine, Vol. 205, No. 7, 1517-1522
The Rockefeller University Press, 0022-1007 $30.00
© 2008 Steinman
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COMMENTARY

 A rush to judgment on Th17

Lawrence Steinman

L.S. is at Department of Neurology and Neurological Sciences and the Interdepartmental Program in Immunology, Beckman Center for Molecular Medicine, Stanford University, Stanford, CA 94305

CORRESPONDENCE L.S.: steinman{at}stanford.edu


ABSTRACT
Some immunologists have characterized T helper (Th)17 T cells as the master mediators of tissue damage in a variety of pathological conditions. New data now demonstrate that Th1 and Th17 T cells are independently capable of inducing disease in two established models of autoimmunity. Thus, the role of Th17 cytokines as the central mediators of pathological tissue damage seems to require clarification.


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